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Fig. 5 | BMC Complementary and Alternative Medicine

Fig. 5

From: Bioactive components and mechanisms of Chinese poplar propolis alleviates oxidized low-density lipoprotein-induced endothelial cells injury

Fig. 5

EACP activated PI3K/Akt and depressed LOX-1/ p38 MAPK in ox-LDL induced HUVECs at 6 h. a, Expression of Akt, p-Akt, PI3K, p-PI3K after treatment with EACP in ox-LDL induced HUVECs at 6 h, respectively; b, Quantification of relative expression quantity of p-Akt and p-PI3K in ox-LDL induced HUVECs at 6 h, respectively. c, Expression of p-Akt, p-mTOR, full length PARP after treatment with EACP combined with wortmannin. d, Quantification of relative expression quantity of p-mTOR and full length PARP after treatment with EACP combined with wortmannin. e, Expression of LOX-1, p38 MAPK, p-p38 MAPK after treatment with EACP in ox-LDL induced HUVECs at 6 h, respectively; f, Quantification of relative expression quantity of LOX-1 and p-p38 MAPK in ox-LDL induced HUVECs at 6 h, respectively. (*P < 0.05, **P < 0.01 vs ox-LDL group, n = 3). Data are means ± S.E.M.

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