Fig. 6From: Ethanol extract of propolis and its constituent caffeic acid phenethyl ester inhibit breast cancer cells proliferation in inflammatory microenvironment by inhibiting TLR4 signal pathway and inducing apoptosis and autophagyEECP and CAPE regulated the levels of TLR4, MyD88, IRAK4 and TRIF. a Cells were treated with EECP and CAPE for 24 h, respectively. Cells were stained with anti-TLR4 antibody. Immunofluorescence graphs showed a decrease of TLR4 level. b The expression of TLR4, MyD88, IRAK4 and TRIF in LPS-stimulated MDA-MB-231 cells were detected by western blotting at 48 h. c Quantification of relative expression of TLR4, MyD88, IRAK4 and TRIF in LPS-stimulated MDA-MB-231 cells. (* P < 0.05, ** P < 0.01 vs control, n = 3). Data are means ± S.E.MBack to article page